Regulation of Cardiac L-Type Ca2+ Current in Na+-Ca2+ Exchanger Knockout Mice: Functional Coupling of the Ca2+ Channel and the Na+-Ca2+ Exchanger

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Transport and Regulation of the Cardiac Na+-Ca2+ Exchanger, NCX1

Cardiac muscle fails to relax upon replacement of extracellular Ca2+ with Ba2+. Among the manifold consequences of this intervention, one major possibility is that Na(+)-Ba2+ exchange is inadequate to support normal relaxation. This could occur due to reduced transport rates of Na(+)-Ba2+ exchange and/or by failure of Ba2+ to activate the exchanger molecule at the high affinity regulatory Ca2+ ...

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ژورنال

عنوان ژورنال: Biophysical Journal

سال: 2007

ISSN: 0006-3495

DOI: 10.1529/biophysj.106.091538